Metabolic Effects of Critical Illness
Metabolic Effects of Critical Illness
David Ray Velez, MD
Catabolism
- Catabolism Predominates in Critical Illness Causing Breakdown of Glycogen, Protein, and Fat
- Glucose is the Preferential Substrate in Critical Illness
- Liver Produces Large Amounts of Glucose Causing a Stress Hyperglycemia
Glucose Production
- Glycogenolysis: Glycogen Store Breakdown to Glucose
- Stores are Rapidly Used/Depleted Early in the Stress Response
- Muscle (2/3 of Stores) – Only Available Locally
- Liver (1/3 of Stores) – Available Systemically
- Gluconeogenesis: Glucose Creation from Non-Glycogen Precursors
- Produced from Alanine, Glycerol, and Lactate (Cori Cycle) in the Liver (Primarily) and Kidney
- Proteolysis: Muscle Proteins Breakdown to Amino Acids (Alanine and Glutamine) for Gluconeogenesis
- Causes Muscle Wasting – Lose 1-5% of Lean Body Mass Per Day (ICU-Acquired Weakness)
- Significantly Negative Nitrogen Balance
- Lipolysis: Fat/Lipid Breakdown to Fatty Acids for Energy Production
- Lipids are Used Less than Carbohydrates – Lipid Conversion to ATP Requires Significant Oxygen in Mitochondria (Often Unavailable in Critical Illness)
Insulin Resistance
- Can Develop During Critical Illness Even without Preexisting Diabetes
- Causes:
- Stress Response Stimulating Cortisol and Catecholamine Release
- Systemic Inflammation Impairing Insulin Effects
- Tissue Injury
- Malnutrition
- Hepatic Glycogenolysis and Gluconeogenesis are Overactivated
- Insulin-Driven Carbohydrate/Glucose Delivery to Peripheral Tissues is Not a Priority
- Can Exacerbate Hyperglycemia and Increase Morbidity and Mortality
Phases
- Ebb Phase (8-24 Hours): Dominated by Circulatory Changes Requiring Resuscitation
- Catabolic Phase (3-10 Days): Dominated by Catabolism Due to Cytokine Mediators (IL-6)
- Anabolic Phase (Recovery): Dominated by Anabolism with Repair and Synthetic Activity
ICU-Acquired Weakness
- Muscle Loss Starts within 4 Hours of Being Immobilized in the ICU
- 1% of Muscle Mass is Lost Every Day of Immobilization
- Risk Factors:
- Prolonged Bedrest or Immobilization
- Severity of Illness (Sepsis, SIRS, Multiple Organ Failure, etc)
- Hyperglycemia
- TPN
- Elderly
- Female Sex
- Premorbid Obesity
- Steroids
- Sedatives or Paralysis
- Pathogenesis is Multifactorial
- Increases Risk of Infections, Dysphagia, Prolonged Mechanical Ventilation, LOS, and Mortality